Restore Movement and Control After Paralysis
Is Paralysis Limiting Your Life?
Paralysis is the loss of voluntary movement that happens when communication between the brain, spinal cord, and muscles is disrupted. For some patients, muscles become stiff and tight, making every movement feel like a struggle; for others, the muscles are weak, floppy, and unable to respond at all.
These changes affect more than motion. They influence how you work, care for yourself, interact with loved ones, and move through the world. The goal of our Center is to pinpoint exactly where the communication breakdown has occurred and build a targeted plan that addresses not only the nerves, but also the muscles and joints involved.
You do not have to navigate this alone. Here’s who we help:
Patients with spastic paralysis after stroke, cerebral palsy, traumatic or anoxic brain injury, or certain spinal cord injuries.
Patients with flaccid paralysis after brachial plexus injury, peripheral nerve injury, or select spinal cord injuries.
Patients with a mixed pattern, where some muscles are stiff and others are weak or floppy in the same limb.
Children and adults who have tried therapy, bracing, and injections but still live with deformity, loss of function, or caregiving challenges.
People whose paralysis has led to joint contractures, limb deformity, or difficulty with positioning, hygiene, and daily care.
Types of Paralysis We Treat
Our specialists diagnose and treat a wide spectrum of paralytic conditions that are often managed piecemeal elsewhere. Here, they are brought together under one coordinated surgical and rehabilitation program.
Spastic Paralysis
Spastic paralysis occurs when the upper motor neurons in the brain and upper spinal cord are damaged. These neurons normally act as a command center and braking system—planning movement and keeping reflexes under control. When they are injured, muscles become overactive and stiff, leading to spasticity, progressive muscle shortening, and joint contractures.
Learn more about:
- Spastic Paralysis – Overview of causes, symptoms, and treatment options.
- Stroke (CVA) – Spasticity and limb deformity after stroke.
- Aneurysm Rupture and Brain Bleeds – Spastic paralysis after intracranial hemorrhage.
- Traumatic Brain Injury / Anoxic Brain Injury – Movement problems following head trauma or oxygen loss.
- Cerebral Palsy – Lifelong spasticity affecting upper and/or lower extremities.
- Upper Extremity Spasticity – Stiff shoulders, elbows, wrists, and hands.
- Lower Extremity Spasticity – Equinus, crouch, or scissoring gait patterns.
- Facial Spasticity – Involuntary facial contractions and abnormal positioning.
Flaccid Paralysis
Flaccid paralysis occurs when the lower motor neurons—the peripheral nerves and roots that carry signals from the spinal cord to the muscles—are damaged. Without this final link in the chain, muscles become weak, soft, and unresponsive, and over time they may shrink due to lack of use.
Learn more about:
- Flaccid Paralysis – Overview of causes, symptoms, and reconstruction options.
- Brachial Plexus Injury – Paralysis of the shoulder, arm, and hand after trauma or stretch.
- Peripheral Nerve Injury – Nerve cuts, crush injuries, and compressions in the limbs.
- Tendon/Muscle Injury – Loss of function from tendon rupture or muscle damage, sometimes combined with nerve injury.
- Spinal Cord Injury – Mixed spastic and flaccid patterns depending on level and severity.
- Wrist Drop, Foot Drop, Facial Nerve Palsy – Focal paralysis affecting key movements.
How Paralysis Develops and Why Timing Matters
Paralysis is not static. Without the right interventions, it follows a predictable path that can make treatment more complex over time.
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In spastic paralysis, loss of upper motor neuron control removes the brain’s braking system. Reflexes become overactive, leading to increased tone, muscle shortening, and eventually fixed joint contractures if not addressed.
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In flaccid paralysis, loss of nerve signal prevents muscles from contracting. Muscles weaken and atrophy, and joints can become stiff if they are not moved regularly.
Our team uses a structured progression model to guide treatment:
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Increased tone → addressed with nerve‑based procedures and injection management.
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Muscle shortening → addressed with muscle or tendon lengthening and focused therapy.
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Joint contracture → addressed with joint release procedures when the joint itself is stiff.
By treating only what is present—nerve, muscle, joint, or all three—we avoid overtreatment while still fully addressing the functional problem.
Advanced Procedures That Relieve Chronic Pain
These surgical options are always paired with intensive therapy before and after surgery to maintain motion, retrain movement patterns, and protect the reconstruction.
Why Patients Choose The Institute’s Paralysis Center
The Center for Surgical Treatment of Paralysis brings together a team uniquely equipped to manage complex spastic and flaccid paralysis:
Performed
Studies Published
Served
Microsurgical Techniques
Want to know how our breakthroughs translate into better outcomes?
The Critical Role of
Therapy and Physiatry
Surgery is only one part of successful paralysis care. Physical and occupational therapy, guided by a physiatrist, are essential at every step.
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Therapy helps prevent joint stiffness, reduce muscle shortening, maintain mobility, and retrain the brain and muscles to use new nerve pathways after surgery.
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Custom splints and braces position limbs for comfort and function and protect surgical corrections during healing.
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Physiatry (PM&R) provides expert direction for therapy plans, prescribes medications, and offers targeted Botox or phenol injections to manage spasticity when appropriate.
We often describe joints as ‘hinges on a door’—they must keep moving to stay healthy, or they risk becoming stiff and limiting future reconstruction. Before we reanimate a limb with nerve or muscle surgery, we make sure those hinges are not “rusty.”
Meet Our Paralysis Surgeons
Behind every successful reconstruction is a team that understands the complexity of nerve injury and the impact of paralysis on daily life. Our surgeons in the Center for Surgical Treatment of Paralysis offer options that are not widely available elsewhere.
Andrew Elkwood
Hamid Abdollahi
Peter Andrawes
Beth Anglin
Gary Arishita
Russell Ashinoff
Sean Boutros
Anjeanette Brown
David Cabiling
Courtney Carpenter
Stephen Chagares
Eric I. Chang
Ann Chuang
Frank Ciminello
Kari L. Colen
Ryan Couvillion
Desiree D'Angelo
Joseph Dayan
Frank Dellacroce
George Dreszer
Stephen Dudick
Rodion Erenburg
Berry Fairchild
Adam Feintisch
Nicole Ferro
James Fletcher
Anton Fries
James Frost
Amon-Ra Gama
Andrew Gassman
Patrick Greaney
Ritwik Grover
Edward Hahn
Sanaz Harirchian
Cassie Hartline
Sean Herman
Zuhaib Ibrahim
Lisa Iucci
Hari Iyer
Priya Jadeja
Reza Jarrahy
Santosh Kale
Karen Kaplan
Matthew Kaufman
Ergun Kocak
Arjuna Kuperan
Chris Lakhiani
Peter Ledoux
Matthew Lynch
Tyler Merceron
Ahmed Nasser
Chet Nastala
Robert T. Nevitt III
Oscar Ochoa
William Ordoyne
Lauren Otaguro
Tushar Patel
Steven Pisano
Vaishali Purohit
Sidney Rabinowitz
Deepak Ramesh
David Rayfield
Rukmini Rednam
Michael Rose
Adam Saad
Nirvana Saraswat
Ali Seckin
Ajul Shah
Tzvi Small
Mohit Sood
Scott Sullivan
Yolanda Tammaro
Pankaj Tiwari
Stephen Toran
Christopher Trahan
David Tsai
Hakan Usal
Eric Weiss
Jonathan Weiswasser
Lauren Whipple
Eric Wimmers
Whitten Wise
Mary Jo Wright
Alex Xu
Mark Yazid
Sarosh Zafar
Your Questions, Answered
Our team evaluates muscle tone, reflexes, strength, and joint motion to determine whether your paralysis is spastic, flaccid, or mixed. This distinction guides whether we focus on nerve‑based procedures to reduce tone, nerve transfers to restore signal, or a combination of both.
→ Learn more on our Spastic Paralysis and Flaccid Paralysis pages.
Botox and phenol injections can temporarily relax overactive muscles and improve motion, but their effects wear off and repeat treatments are often required every few months. For many patients, these injections are part of a broader plan that may eventually include surgery.
→ Visit our Spastic Paralysis page to explore injection and surgical options.
We consider surgery when spasticity or weakness is causing significant functional limitations, deformity, or caregiving challenges despite optimized therapy, bracing, and injections. Timing is especially important for nerve transfers, which work best before muscles have been without signal for too long.
Over time, untreated spasticity can lead to muscle and joint contractures, while longstanding flaccid paralysis can result in irreversible muscle atrophy and joint stiffness. Earlier evaluation allows us to preserve more options and potentially achieve better outcomes.
→ Learn more about why patients choose us.
What to Expect When You Reach Out
We know reaching this point has likely been a long journey. That’s why we make the next step clear and supportive.
1 Consultation
2 Evaluation
3 Personalized Care Plan
4 Surgery & Support
Get Relief From Paralysis‑Related Pain and Limitations
Paralysis shouldn’t define what you can do each day. At the Center for Surgical Treatment of Paralysis at The Institute for Advanced Reconstruction, our team combines advanced nerve and muscle surgery with specialized rehabilitation to help you regain movement, comfort, and control.
Ready to explore your options?